Friday, November 30, 2012

Atheist Ethicist: Moral Hazard: Bail outs, Debt relief, and Immigration

Our last conversation brought up the subject of moral hazard.

This is a term largely used in economics. There, it is typically used to refer to policies that remove the costs of failure - thus giving people an incentive to take risks they would not otherwise take. These risks end up costing society a great deal, because they have to cover the cost of failure.

As an illustrative example, imagine a game of poker. One of the players receives a promise from an observer, "If you win, you keep all of your winnings. But if you lose, I will cover all of your losses."

This creates a situation where the player now a huge and perverse incentive to take all sorts of risks he would not otherwise take. He has an incentive to try for "long shots" - plays that pay off big if they succeed, but almost never succeed.

How many lottery tickets would you buy if somebody said, "You can buy as many as you want. I will cover the cost of every ticket that does not win?"

Now, the person making the promise is the government. The people they make this promise to are investors - your standard "Wall Street Bankers". Propagandists tell us that the government spends too much money helping the poor and middle class. Yet, huge amounts of government money go to helping the wealthy avoid major losses - helping the very people who refuse to any taxes to cover these guarantees.

"Moral Hazard" is tightly linked to "To Big To Fail". The reason the government covers the losses of these risk takers is because of the costs of failure to the economy as a whole. By knowing that the government cannot possibly allow these costs to stand, the government does not even need to explicity cover these costs. Thus those companies take huge risks, they fail, and the bailout begins (bailouts that those who are bailed out are refusing to pay for).

I have described this with respect to multi-billion dollar government bailouts. It applies to regular borrowers as well.

Imagine two households. Household 1 purchases a $100,000 house, refrains from buying expensive gadgets or vacations, saves for retirement, and keeps their debt manageable. The value of their house goes up over the next several years, but they allow the equity to build and maintain their current life style. After 10 years, the value of their house collapses back down to $100,000. However, they now owe $50,000, which they can easily continue to cover.

Household 2 buys a $200,000 house. As housing prices rise they refinance and spend the equity on cruises or other forms of entertainment and gadgets. At the end of 10 years, the value of their house collapses back down to $200,000. However, they have $300,000 worth of mortgages from refinancing. At this point, the government steps in to give them assistance with their loan. This household ends up after 10 years with a $200,000 house, a house full of gadgets, and memories of the places they have seen and the things they have done.

Now, to add injury to insult, the government needs money to cover these costs. It can only get the money from those who managed their finances responsibility and, consequently, have money to spare. The person who gave up all sorts of luxuries and who kept his finances in order finds himself with an additional tax burden precisely because he has to give some of his money to the household that spent wildly.

The moral hazard comes from the fact that such a policy rewards (in the biological sense) those who are financially irresponsible and punishes those who are financially responsible. It teaches a lesson that those who spend wildly and accumulate massive debts enjoy a greater quality of life over the long run than those who manage their finances responsibly. This, in turn, sets the stage for yet another round of fiscal irresponsibility - one in which people have been taught to sense the rewards of being one of those who act irresponsibility and sense the costs of being responsible.

I should add that it is not the case that all people who end up in financial distress have mismanaged their money. They might end up in this situation due to a severe illness (though illnesses brought about by poor life-style choices such as drinking, smoking, and obesity will not count in this regard). Criminals might take a person's ability to pay their debts, or some (unforeseeable) natural disaster (against which proper precautious could not have been taken) might have caused them great harm. However, there are people who end up in financial distress due to their own actions.

In that previous discussion I mentioned at the start of this article, moral hazard came into play regarding immigration reform.

Let us again take a situation that involves two people in another country in identical circumstances. The one difference between these two people is that one has a disregard for the law or the rules. He does as he pleases and tries to get away with what he can. He crosses into the country illegally and gets a job. The other person has a respect for the rules. He learns and tries to follow all of the proper procedures. However, this involves a lot of red tape and waiting with no guarantee of acceptance, so he remains out of the country legally.

Now, an amnesty is declared. In doing so, the person with low respect for the law and regard for the rules ends up getting a significant advantage - he ends up being accepted into this country. On the other hand, the one who respected and followed the rules is kept out. In fact, his chances of getting into the country may be reduced because the "quotas" are taken up by those who came into the country illegally.

Here, we have created a situation where we have rewarded (in the biological sense) disregard for the law and a willingness to do what one wishes, and punished (in the biological sense) those who are inclined to follow the rules and accomplish their ends legitimately. This, in turn, sets the stage for yet another round of illegal immigration - one in which more people see the advantages of breaking the law and hoping for the next amnesty, and fewer people see any reason to respect the rules and procedures that have been put into place regarding immigration.

All of these elements of moral hazard are legitimate.

In practice, we tend to see Republicans who ignore the moral hazard of "too big to fail" government bailouts - or even benefit from the government's implicit promise of future bailouts - by paying any additional taxes and fees to the government. Those practitioners are permitted to keep everything that they get when they win, while having others cover their debts when they lose. While debates go on about funding the massive deficit that, to a substantial degree, was created to bail out these people, we hear them demanding that they should pay nothing. They should only obtain government benefits - and never pay the costs.

At the same time we see Democrats that ignore the moral hazard of rewarding fiscal irresponsibility and a disregard for the rules on the part of the middle class and poor.

In fact, moral hazard is a legitimate concern - a legitimate reason for action - at all levels. What we should be doing is creating institutions that reward responsibility and respect for the rules, while at least forcing people to accept the costs of their own failures and preventing them from obtaining benefits through criminal activity.

Source: http://atheistethicist.blogspot.com/2012/11/moral-hazard-bail-outs-debt-relief-and.html

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Syrian Man Helps Disaster Relief Organization Distribute Aid to Fellow Refugees in Iraq

After fleeing the military he was forced to join two years ago, a Syrian man has become invaluable to the volunteers from international disaster relief organization, ShelterBox, as they delivered aid to thousands of other refugees living in the Domiz camp in Iraq Kurdistan.

Sarasota, Florida (PRWEB) November 28, 2012

After fleeing the military he was forced to join two years ago, a Syrian man has become invaluable to the volunteers from international disaster relief organization, ShelterBox, as they delivered aid to thousands of other refugees living in the Domiz camp in Iraq Kurdistan.

ShelterBox responds immediately to disasters such as earthquake, volcano, flood, hurricane, cyclone, tsunami or conflict, by delivering boxes of aid. Each box includes a disaster relief tent, water purification kit, cook-stoves, blankets and other supplies that help families survive and rebuild their lives in the days, weeks or months following disaster.

Members of the highly trained ShelterBox Response Team (SRT) were delivering boxes of winterized shelter and supplies to families living in the camp when they met Abdullah, a man who fled the Syrian Army after being forced to fight his own people.

David Webber, an SRT member from the UK, said they first met Abdullah when they were setting up the first shipment of tents. He noticed that Abdullah spoke English quite well, and asked if he would act as the SRT?s guide and translator during their deployment.

?Without [Abdullah], our work here would not have been as quick and efficient ? we now have 500 tents set up, bringing shelter, warmth and dignity to families,? Webber said.

Abdullah was taken from his home in Qamishli, Syria, in October 2010. He was forced to quit studying English at the University of Aleppo and instead join the soldiers fighting for President Bashar al-Assad.

In June 2012, he escaped the growing violence between military troops and rebel fighters when he crossed the border into Iraq. He has since been living in the Domiz camp with his aunt, uncle, nieces and nephews.

?Meeting [volunteers] from ShelterBox has been amazing for all of us here,? Abdullah said. ?Not only do I have work to provide for my family here, but we are also helping my people who are in desperate need by bringing them what's most important to them at this time ? safety and hope.?

ShelterBox is also responding in the aftermath of Hurricane Sandy in Haiti, to earthquakes in Guatemala, and extensive flooding in Nigeria, Senegal and Uganda.

ABOUT SHELTERBOX USA


Since 2000, ShelterBox has provided shelter, warmth and dignity following more than 200 disasters in over 85 countries. ShelterBox instantly responds to earthquake, volcano, flood, hurricane, cyclone, tsunami or conflict by delivering boxes of aid. Each iconic green ShelterBox contains a disaster relief tent for an extended family, stove, blankets and water filtration system, among other tools for survival. ShelterBox?s American affiliate, ShelterBox USA is headquartered in Sarasota, Florida. Individual tax-deductible donations to ShelterBox USA can be made at http://www.shelterboxusa.org, 941-907-6036 or via text message by sending SHELTER to 20222 for a one-time $10 donation.


# # #

Tiffany Stephenson
ShelterBox USA
(941) 907-6036 102
Email Information

Source: http://news.yahoo.com/syrian-man-helps-disaster-relief-organization-distribute-aid-181630185.html

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How Amazingly Convenient: Lefties 'Discover' That Susan Rice Has 'Conflict of Interest' Energy Investments

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Source: newsbusters.org. --- Wednesday, November 28, 2012
In what would appear to be a sure sign that the Obama administration's leftist allies, perhaps with the President's go-ahead, are preparing to throw current U.N. ambassador Susan Rice under the bus, Alex Guillen at the Politico reported at 6:14 p.m. on information that has from all appearances been public information for at least three months , but which the National Resources Defense Council's On Earth blog ?noted about an hour earlier. Rice's offenses? She "holds significant investments in more than a dozen Canadian oil companies and banks that would stand to benefit from expansion of the North American tar sands industry and construction of the proposed $7 billion Keystone XL pipeline." That's indeed troubling, but it was just as troubling when leftists up to and including the editorialists at the Washington Post were accusing anyone objecting to Rice's potential nomination of being presumptively racist. Excerpts from Guillen's report follow the jump (bolds are mine): read more ...

Source: http://newsbusters.org./blogs/tom-blumer/2012/11/28/how-amazingly-convenient-lefties-discover-susan-rice-has-conflict-intere

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40th Annual UBS Global Media and Communications Conference ...

NEW YORK -?The annual UBS Global Media and Communications Conference is one of the longest running conferences on Wall Street. The conference will provide investors with a comprehensive look at the media and communications space, recap 2012, and also provide insights on the years ahead.

Some of companies presenting during the conference include: DirecTV, Netflix, Time?Warner Cable, The Walt Disney Company, CBS Corporation, IMAX Corporation, and Roku Inc.

The 40th?annual UBS Global Media and Communications Conference taking place from December 3-5, 2012 in New York.?Michael Donovan, chief executive officer of DHX Media, will present to investors on?December 5?at?9:30am EST.

Continuing its strategy of delivering exceptional creative content to audiences around the world, The Walt Disney Company (NYSE: DIS) agreed to acquire Lucasfilm Ltd. in a stock and cash transaction. Lucasfilm is 100% owned by Lucasfilm Chairman and Founder, George Lucas.

Under the terms of the agreement and based on the closing price of Disney stock on October 26, 2012, the transaction value is $4.05 billion, with Disney paying approximately half of the consideration in cash and issuing approximately 40 million shares at closing. The final consideration will be subject to customary post-closing balance sheet adjustments.

?Lucasfilm reflects the extraordinary passion, vision, and storytelling of its founder, George Lucas,? said Robert A. Iger, Chairman and Chief Executive Officer of The Walt Disney Company. ?This transaction combines a world-class portfolio of content including?Star Wars,?one of the greatest family entertainment franchises of all time, with Disney?s unique and unparalleled creativity across multiple platforms, businesses, and markets to generate sustained growth and drive significant long-term value.?

?For the past 35 years, one of my greatest pleasures has been to see?Star Wars?passed from one generation to the next,? said George Lucas, Chairman and Chief Executive Officer of Lucasfilm. ?It?s now time for me to pass?Star Wars?on to a new generation of filmmakers. I?ve always believed that?Star Wars?could live beyond me, and I thought it was important to set up the transition during my lifetime. I?m confident that with Lucasfilm under the leadership of Kathleen Kennedy, and having a new home within the Disney organization,?Star Wars?will certainly live on and flourish for many generations to come. Disney?s reach and experience give Lucasfilm the opportunity to blaze new trails in film, television, interactive media, theme parks, live entertainment, and consumer products.?

?

Jay Rasulo, Senior Executive Vice President and Chief Financial Officer, The Walt Disney Company, will participate in a question-and-answer session at the?40th Annual UBS Global Media and Communications Conference on Wednesday, December 5, 2012 at approximately 12:15 p.m. EST / 9:15 a.m. PST.

DHX Media (www.dhxmedia.com) is a leader in the creation, production and licensing of family entertainment rights. DHX Media owns, markets and distributes over 8,500 half hours of children?s entertainment content, and exploits owned properties through its consumer products licensing business. DHX Media is? recognized for? brands such as?Caillou,?Richard Scarry?s Busytown Mysteries,?Inspector Gadget,?Johnny Test,?Animal?Mechanicals,?Kid vs. Kat,?Super WHY!,?Rastamouse, and?Yo Gabba Gabba!. The company also provides programming for Cookie Jar TV, the weekend morning block on CBS. DHX Media?s full-service international licensing agency, Copyright Promotions Licensing Group, (CPLG), represents numerous entertainment, sport and design brands. DHX Media has offices in?Toronto,?Los Angeles,?Vancouver,?Halifax,?London,?Paris,?Barcelona,?Lisbon,?Milan,?Munich,Netherlands?and is listed on the?Toronto?Stock Exchange.

Source: DHX MEDIA LTD.

Source:?thewaltdisneycompany.com/

Short URL: http://woodlawnpost.com/?p=67924

Source: http://woodlawnpost.com/?p=67924&utm_source=rss&utm_medium=rss&utm_campaign=40th-annual-ubs-global-media-and-communications-conference-december-3rd-5th-2102

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Biggio on the ballot (Offthekuff)

Share With Friends: Share on FacebookTweet ThisPost to Google-BuzzSend on GmailPost to Linked-InSubscribe to This Feed | Rss To Twitter | Politics - Top Stories News, News Feeds and News via Feedzilla.

Source: http://news.feedzilla.com/en_us/stories/politics/top-stories/267155280?client_source=feed&format=rss

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Thursday, November 29, 2012

FreedomWorks and Pennsylvania Small Business Owners to Hold ...

Warminster, PA- FreedomWorks and a group of local small business owners in the Bucks County area will hold a press conference to express concerns over the economic damage at risk if President Obama raises taxes on America?s small businesses.

Speakers will include:? Vice President of the Iron League Dan Beisel, President of the PA Chamber of Business and Industry Gene Barr, FreedomWorks PA State Director Ana Puig, President of American Bar Products Inc. William Marsh, Executive Director of PA Manufacturers? Association David Taylor and PA State Director of the National Federation of Independent Business Kevin Shivers.

WHEN: Friday November 30, 2012 from 2-2:30pm ET.
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WHERE: American Bar Products, 971 Mearns Road, Warminster, PA 18974.

WHY:? The purpose of the press conference is to raise awareness of the current state of manufacturing in Pennsylvania due to over-taxation and over-regulation, and how the economic policy coming out of the fiscal cliff negotiations will affect the ability of small businesses to create jobs.

FreedomWorks and its Pennsylvania membership urge Congress and the White House to extend the current tax rates, make the promised $1.2 trillion in spending cuts and to put responsible solutions on the table for fundamental tax and entitlement reform. For more information about FreedomWorks, please visit www.FreedomWorks.org.

Source: http://www.freedomworks.org/press-releases/freedomworks-and-pennsylvania-small-business-owner

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Stop Touching Yourself, Flu Researchers Say

During cold and flu season, we're inundated with messages to wash our hands frequently. But to avoid getting sick, it's also important that we stop touching our noses and mouths all the time, a new study shows.

Every time people touch their mouth or nose, they transfer bacteria and viruses between their face and their hand. This "self-inoculation," or transfer of germs from one body part to another, is a primary way that germs wind up spreading from contaminated surfaces to people's faces, and from sick people to often-touched surfaces.

"There are many opportunities in between hand-washing episodes for people to re-contaminate their hands," said study researcher Dr. Wladimir Alonso, a global health researcher at the National Institutes of Health in Bethesda, Md.

Alonso and colleagues randomly selected 249 people in public places,?on the Washington, D.C. subway and in the Brazilian city of Florianopolis. The researchers?observed them, noting how often they touched a common surface and then their mouth or nose. They found that people touched their faces an average of 3.6 times per hour, and common objects an average of 3.3 times per hour.

This rate of self-touching means that people likely get germs on their hands much more frequently than they wash germs off their hands, according to the study.

"It is important to understand the basic mechanisms through which diseases are transmitted to take full advantage," of hand-washing, Alonso said.

Recommendations issued to the public typically emphasize hand-washing, but during potentially severe disease outbreaks, the messages should be shifted to ensure that people understand how self-inoculation occurs, and avoid touching their faces, the researchers said in their study.

"If a deadly respiratory virus is around, this is something to really take into account," Alonso said, pointing to the 2009 flu pandemic?as one example of a situation where knowledge of self-inoculation could limit the spread of disease.

Alonso said that knowing how often self-inoculation happens should not turn people into hypochondriacs, or toward a life of discomfort and constant state of alert. The immune system offers good protection against diseases.

"But it is also important to be aware that re-contamination can occur very quickly after we wash our hands," he said.

The findings were published Nov. 15 in the journal Clinical Infectious Diseases.

Pass it on: Touching your mouth or nose can spread the flu.

FollowMyHealthNewsDaily?on Twitter?@MyHealth_MHND. We're also on?Facebook?&?Google+.

Copyright 2012 MyHealthNewsDaily, a TechMediaNetwork company. All rights reserved. This material may not be published, broadcast, rewritten or redistributed.

Source: http://news.yahoo.com/stop-touching-yourself-flu-researchers-173421678.html

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ICANN, Make a Difference

Internet cafe in Nairobi A cybercafe in the Kibera slum in Nairobi

Photo by TONY KARUMBA/AFP/GettyImages.

This article arises from Future Tense, a joint effort of Arizona State University, the New America Foundation, and Slate that looks at emerging technologies and their implications for policy and for society. On Thursday, Nov. 29, Future Tense will host an event in Washington, D.C., on the future of Internet governance. To learn more and to RSVP, visit the New America Foundation?s website. The event will also be streamed.

The Internet Corporation for Assigned Names and Numbers is little known, but it wields a tremendous amount of power: It controls all of the Web?s top-level domains (those letters after the ?dot,? like .com and .org). Currently, ICANN is in the midst of creating hundreds (and possibly thousands) of new, generic top-level domains (gTLDs) that span a host of different ideas, from .web to .cars to .anything_else_you_can_imagine. These new gTLDs have the potential to dramatically affect the future of Internet browsing, and they?re already stirring up some serious discussion. (Saudi Arabia, for one, doesn?t want .gay, .bible, or other dozens of other proposed domains to be approved.) But the auction process to distribute them also has the potential for even greater impact than currently envisioned.

ICANN?s new generic top-level domain process has been dragging on for years?basically since the organization?s inception in 1998. But this year, it is finally coming to fruition, and as early as April 2013 we are likely to see the first group of new gTLDS?in essence, ICANN will empower specific legal entities to control how to use and sell these domain names. This process means significant amounts of money may start rolling in soon: It already costs $185,000 to apply for a gTLD, but when there are multiple bidders for the same string?like .web or .app?they will be put up for auction.

For instance, the .web gTLD is widely desired by a number of different organizations, as it is the most likely contender to possibly challenge the king of all gTLDs: .com. There are currently eight applicants for .web, including Google, German Internet giant 1&1, and incumbent registry operator Afilias (which manages .org and .info) among other bidders. We expect that the bidding for .web alone is likely to be in excess of $5 million and could potentially reach $10 million or more. For the .app gTLD, there are 11 applicants?and we may see a titanic bidding war between Google and Amazon. There are hundreds more contended strings that are likely to go to auction and raise tens of millions of additional dollars?even $100 million isn?t out of the question.

All told, there are more than 1,900 applications for roughly 1,000 unique strings in this first wave. The $185,000 application fee is intended to fund the ICANN process, but the proceeds from contention auctions are considered ?excess funds? that are not already earmarked to cover costs. The challenge will be to use these proceeds in a way that best benefits the public interest and the global Internet. In talking with key stakeholders over the last couple of years, everyone agrees that allocating these funds will be a challenge and likely to be fraught with politics.

We've been involved in the ICANN process since its inception, and believe that these proceeds can and should be used to do something game-changing and truly visionary: build and maintain free wireless Internet infrastructure for huge swaths of the continent of Africa or an equally disconnected, high-poverty area of the planet. This is an audacious idea that many might originally dismiss as impractical?but that's because their thinking is stuck inside the box. We know that it can be done?and how. Providing free wireless Internet infrastructure for the continent of Africa would be a dream come true?the kind of outcome that would help bridge the digital divide and garner huge socioeconomic benefits for decades to come.

But would $100 million actually be enough to build useful, sustainable infrastructure? It would, if its creators use many of the newer ?mesh? technologies that are now coming online. African's Internet penetration currently hovers around 15 percent?less than half the world average. And roughly half of African countries have single-digit broadband penetration rates. The digital divide looms large, in no small part because many Africans simply cannot afford current broadband prices.

To help solve this problem, we can use what are called mesh technologies, which are significantly cheaper than the systems we use in the developed world. More importantly, they can easily interconnect and extend existing infrastructure in remarkably cost-effective ways. Instead of connecting people through a central hub or tower, mesh architectures resemble more of a spider web, in which data can flow through a large number of routes to reach their destination. Because there's no single point of failure within a mesh network and because of the redundancy of pathways within these systems, they're often more resilient?harder for human intervention or natural disaster to take down as well as more difficult to surveille and censor.

Projects like CommotionWireless.net and OpenGarden.net have developed game-changing technologies that allow existing cellphones to connect with each other directly, for free. They can also share bandwidth from one cellphone throughout an entire network of devices, allowing people who don't have Internet connectivity to browse the Web or send email via someone else's connection. Now what's needed is a bold, widespread implementation of these technologies. There's no reason Africa shouldn?t take advantage of new technologies and new business models to provide a modest amount of connectivity each month for anyone who wants it.

In essence, what we're proposing is the widespread unlocking of existing technologies, the creation of hybrid networks that take advantage of recent advances in ad-hoc wireless networking, and the digital enfranchisement of the billions of people who cannot afford to participate in current business models. Providing basic connectivity is not expensive?more importantly, unlocking devices and allowing peer-to-peer connectivity costs absolutely nothing.

A $100 million intervention would enable a wide-ranging proof-of-concept of today's cutting-edge mesh wireless technologies. It would demonstrate the viability of new hybrid networking architectures that opportunistically used for-fee services when necessary and offload to free alternatives whenever possible. It would enable us to try out numerous innovative business models. And most importantly, it would point the way forward?helping solve the problem of how to grant access to the Internet's vast resources to the substantial majority of humanity who are not meaningfully online.

With the gTLD auctions now pending, ICANN has a once-in-a-generation opportunity to change the digital destinies of billions of people. With ICANN's help, we can realize a world where Africa rivals any country on the planet when it comes to online connectivity. And where African citizens can take it for granted that meaningful (online) civic participation is universally available. We've already seen how even modest resources like the mobile micropayment system M-Pesa have helped spur a new generation of entrepreneurship?imagine what's possible when free connectivity is available to all.

Source: http://feeds.slate.com/click.phdo?i=f7630f50e0aea65eaa65152bc151252c

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Everything Any individual Needs To Know About Payday Loans ...

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Ensure that you have a real lender?s internet site well before, you add any personal information in. This is significant due to the fact, there are a lot of sites that redirect anyone to other areas. This occasionally imply that you end up obtaining more than one personal loan from separate creditors. You end up owing over you can afford.

When taking out a cash advance, it is important that you do your research. You do not desire to blindly believe an advertisement, simply because you will not be receiving the overall scenario. Speak with people who took out this sort of personal loan, or read a number of the business reviews on the web.

If you sign up for a cash advance, you happen to be definitely taking out the next income as well as burning off several of it. On the flip side, having to pay this price is occasionally essential, to acquire via a small press in life. In any case, understanding is potential. Ideally, this article has motivated anyone to make well informed choices.

Source: http://www.mandarinmnemonics.com/everything-any-individual-needs-to-know-about-payday-loans/

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ScienceDaily: Gene News

ScienceDaily: Gene Newshttp://www.sciencedaily.com/news/health_medicine/genes/ Genes and Genetics News. Read today's medical research in genetics including what can damage genes, what can protect them, and more.en-usTue, 27 Nov 2012 18:53:48 ESTTue, 27 Nov 2012 18:53:48 EST60ScienceDaily: Gene Newshttp://www.sciencedaily.com/images/logosmall.gifhttp://www.sciencedaily.com/news/health_medicine/genes/ For more science articles, visit ScienceDaily.Gene linked to respiratory distress in babieshttp://www.sciencedaily.com/releases/2012/11/121127130256.htm Some infants are more susceptible to potentially life-threatening breathing problems after birth, and rare, inherited DNA differences may explain why, according to new research.Tue, 27 Nov 2012 13:02:02 ESThttp://www.sciencedaily.com/releases/2012/11/121127130256.htmNew understanding of X chromosome inactivationhttp://www.sciencedaily.com/releases/2012/11/121127101534.htm Scientists have broadened our understanding of how cells regulate silencing of the X chromosome in a process known as X-inactivation.Tue, 27 Nov 2012 10:15:15 ESThttp://www.sciencedaily.com/releases/2012/11/121127101534.htmNew mechanism for cancer progression discoveredhttp://www.sciencedaily.com/releases/2012/11/121127094311.htm Researchers have discovered an alternative mechanism for activating rhe oncogene Ras that does not require mutation or hormonal stimulus.Tue, 27 Nov 2012 09:43:43 ESThttp://www.sciencedaily.com/releases/2012/11/121127094311.htmProtein injection points to muscular dystrophy treatmenthttp://www.sciencedaily.com/releases/2012/11/121127094248.htm Scientists have discovered that injecting a novel human protein into muscle affected by Duchenne muscular dystrophy significantly increases its size and strength, findings that could lead to a therapy akin to the use of insulin by diabetics.Tue, 27 Nov 2012 09:42:42 ESThttp://www.sciencedaily.com/releases/2012/11/121127094248.htmChemical 'switches' for neurodegenerative diseases discoveredhttp://www.sciencedaily.com/releases/2012/11/121127093951.htm Researchers have identified and ?switched off? a chemical chain that causes neurodegenerative diseases such as Huntington?s disease, amyotrophic lateral sclerosis and dementia. The findings could one day be of particular therapeutic benefit to Huntington?s disease patients.Tue, 27 Nov 2012 09:39:39 ESThttp://www.sciencedaily.com/releases/2012/11/121127093951.htmMetabolic protein launches sugar feast that nurtures brain tumorshttp://www.sciencedaily.com/releases/2012/11/121126164003.htm PKM2 slips into nucleus to promote cancer; potential biomarker and drug approach discovered.Mon, 26 Nov 2012 16:40:40 ESThttp://www.sciencedaily.com/releases/2012/11/121126164003.htmPossible new treatment for Ewing sarcomahttp://www.sciencedaily.com/releases/2012/11/121126142855.htm Discovery of a new drug with high potential to treat Ewing sarcoma, an often deadly cancer of children and young adults, and the previously unknown mechanism behind it, come hand-in-hand in a new study.Mon, 26 Nov 2012 14:28:28 ESThttp://www.sciencedaily.com/releases/2012/11/121126142855.htmSurvival gene may be key to controlling HIV and hepatitishttp://www.sciencedaily.com/releases/2012/11/121126131349.htm A newly discovered gene that is essential for embryo survival could also hold the key to treating and potentially controlling chronic infections such as HIV, hepatitis and tuberculosis. The gene, called Arih2, is fundamental to the function of the immune system -- making critical decisions about whether to switch on the immune response to an infection.Mon, 26 Nov 2012 13:13:13 ESThttp://www.sciencedaily.com/releases/2012/11/121126131349.htmMicrobial 'missing link' discovered after man impales hand on tree branchhttp://www.sciencedaily.com/releases/2012/11/121126110737.htm Two years ago, a 71-year-old Indiana man impaled his hand on a branch after cutting down a dead tree. The wound caused an infection that led scientists to discover a new bacterium and solve a mystery about how bacteria came to live inside insects.Mon, 26 Nov 2012 11:07:07 ESThttp://www.sciencedaily.com/releases/2012/11/121126110737.htmTransposable elements reveal a stem cell specific class of long noncoding RNAshttp://www.sciencedaily.com/releases/2012/11/121125192838.htm Over a decade after sequencing the human genome, it has now become clear that the genome is not mostly ?junk? as previously thought. In fact, the ENCODE project consortium of dozens of labs and petabytes of data have determined that these ?noncoding? regions house everything from disease trait loci to important regulatory signals, all the way through to new types of RNA-based genes.Sun, 25 Nov 2012 19:28:28 ESThttp://www.sciencedaily.com/releases/2012/11/121125192838.htmNew molecular culprit linked to breast cancer progressionhttp://www.sciencedaily.com/releases/2012/11/121124090511.htm Researchers have uncovered a protein ?partner? commonly used by breast cancer cells to unlock genes needed for spreading the disease around the body. A report on the discovery details how some tumors get the tools they need to metastasize.Sat, 24 Nov 2012 09:05:05 ESThttp://www.sciencedaily.com/releases/2012/11/121124090511.htmNew insights into virus proteome: Unknown proteins of the herpesvirus discoveredhttp://www.sciencedaily.com/releases/2012/11/121123092132.htm The genome encodes the complete information needed by an organism, including that required for protein production. Viruses, which are up to a thousand times smaller than human cells, have considerably smaller genomes. Using a type of herpesvirus as a model system scientists have shown that the genome of this virus contains much more information than previously assumed. The researchers identified several hundred novel proteins, many of which were surprisingly small.Fri, 23 Nov 2012 09:21:21 ESThttp://www.sciencedaily.com/releases/2012/11/121123092132.htmScientists describe elusive replication machinery of flu viruseshttp://www.sciencedaily.com/releases/2012/11/121122152928.htm Scientists have made a major advance in understanding how flu viruses replicate within infected cells. The researchers used cutting-edge molecular biology and electron-microscopy techniques to ?see? one of influenza?s essential protein complexes in unprecedented detail. The images generated in the study show flu virus proteins in the act of self-replication, highlighting the virus?s vulnerabilities that are sure to be of interest to drug developers.Thu, 22 Nov 2012 15:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121122152928.htmProtein folding: Look back on scientific advances made as result of 50-year old puzzlehttp://www.sciencedaily.com/releases/2012/11/121122152910.htm Fifty years after scientists first posed a question about protein folding, the search for answers has led to the creation of a full-fledged field of research that led to major advances in supercomputers, new materials and drug discovery, and shaped our understanding of the basic processes of life, including so-called "protein-folding diseases" such as Alzheimer's, Parkinson's and type II diabetes.Thu, 22 Nov 2012 15:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121122152910.htmStep forward in regenerating and repairing damaged nerve cellshttp://www.sciencedaily.com/releases/2012/11/121121145638.htm Researchers recently uncovered a nerve cell's internal clock, used during embryonic development. This breakthrough could lead to the development of new tools to repair and regenerate nerve cells following injuries to the central nervous system.Wed, 21 Nov 2012 14:56:56 ESThttp://www.sciencedaily.com/releases/2012/11/121121145638.htmArchitecture of rod sensory cilium disrupted by mutationhttp://www.sciencedaily.com/releases/2012/11/121121145621.htm Using a new technique called cryo-electron tomography, scientists have created a three-dimensional map that gives a better understanding of how the architecture of the rod sensory cilium (part of one type of photoreceptor in the eye) is changed by genetic mutation and how that affects its ability to transport proteins as part of the light-sensing process.Wed, 21 Nov 2012 14:56:56 ESThttp://www.sciencedaily.com/releases/2012/11/121121145621.htmAging: Scientists further unravel telomere biologyhttp://www.sciencedaily.com/releases/2012/11/121121130933.htm Researchers have resolved the structure of that allows a telomere-related protein, Cdc13, to form dimers in yeast. Mutations in this region of Cdc13 put the kibosh on the ability of telomerase and other proteins to maintain telomeres.Wed, 21 Nov 2012 13:09:09 ESThttp://www.sciencedaily.com/releases/2012/11/121121130933.htmDrug resistance biomarker could improve cancer treatmenthttp://www.sciencedaily.com/releases/2012/11/121121130811.htm Cancer therapies often have short-lived benefits due to the emergence of genetic mutations that cause drug resistance. A key gene that determines resistance to a range of cancer drugs has been reported in a new study. The study reveals a biomarker that can predict responses to cancer drugs and offers a strategy to treat drug-resistant tumors based on their genetic signature.Wed, 21 Nov 2012 13:08:08 ESThttp://www.sciencedaily.com/releases/2012/11/121121130811.htmGenome packaging: Key to breast cancer developementhttp://www.sciencedaily.com/releases/2012/11/121121130703.htm Two recent studies delve into the role of chromatin modifying enzymes and transcription factors in tumour cells. In one, it was found that the PARP1 enzyme activated by kinase CDK2 is necessary to induce the genes responsible for the proliferation of breast cancer cells in response to progesterone. In another, extensive work has been undertaken to identify those genes activated by the administration of progesterone in breast cancer, the sequences that can be recognized and how these genes are induced.Wed, 21 Nov 2012 13:07:07 ESThttp://www.sciencedaily.com/releases/2012/11/121121130703.htmShort DNA strands in genome may be key to understanding human cognition and diseaseshttp://www.sciencedaily.com/releases/2012/11/121121130643.htm Previously discarded, human-specific ?junk? DNA represents untapped resource in the study of diseases like Alzheimer?s and autism.Wed, 21 Nov 2012 13:06:06 ESThttp://www.sciencedaily.com/releases/2012/11/121121130643.htmBiomarking time: Methylome modifications offer new measure of our 'biological' agehttp://www.sciencedaily.com/releases/2012/11/121121130633.htm In a new study, researchers describe markers and a model that quantify how aging occurs at the level of genes and molecules, providing not just a more precise way to determine how old someone is, but also perhaps anticipate or treat ailments and diseases that come with the passage of time.Wed, 21 Nov 2012 13:06:06 ESThttp://www.sciencedaily.com/releases/2012/11/121121130633.htmKidney tumors have a mind of their ownhttp://www.sciencedaily.com/releases/2012/11/121121104552.htm New research has found there are several different ways that kidney tumors can achieve the same result -- namely, grow.Wed, 21 Nov 2012 10:45:45 ESThttp://www.sciencedaily.com/releases/2012/11/121121104552.htmMechanism to repair clumped proteins explainedhttp://www.sciencedaily.com/releases/2012/11/121121104416.htm Clumped proteins can be dissolved with the aid of cellular repair systems -- a process of critical importance for cell survival especially under conditions of stress. Researchers have now decrypted the fundamental mechanism for dissolving protein aggregates that involves specific molecular chaperones.Wed, 21 Nov 2012 10:44:44 ESThttp://www.sciencedaily.com/releases/2012/11/121121104416.htmNovel mechanism through which normal stromal cells become cancer-promoting stromal cells identifiedhttp://www.sciencedaily.com/releases/2012/11/121121104401.htm New understanding of molecular changes that convert harmless cells surrounding ovarian cancer cells into cells that promote tumor growth and metastasis provides potential new therapeutic targets for this deadly disease, according to new research.Wed, 21 Nov 2012 10:44:44 ESThttp://www.sciencedaily.com/releases/2012/11/121121104401.htmNew test for tuberculosis could improve treatment, prevent deaths in Southern Africahttp://www.sciencedaily.com/releases/2012/11/121120194932.htm A new rapid test for tuberculosis (TB) could substantially and cost-effectively reduce TB deaths and improve treatment in southern Africa -- a region where both HIV and tuberculosis are common.Tue, 20 Nov 2012 19:49:49 ESThttp://www.sciencedaily.com/releases/2012/11/121120194932.htmEvolution of human intellect: Human-specific regulation of neuronal geneshttp://www.sciencedaily.com/releases/2012/11/121120194926.htm A new study has identified hundreds of small regions of the genome that appear to be uniquely regulated in human neurons. These regulatory differences distinguish us from other primates, including monkeys and apes, and as neurons are at the core of our unique cognitive abilities, these features may ultimately hold the key to our intellectual prowess (and also to our potential vulnerability to a wide range of 'human-specific' diseases from autism to Alzheimer's).Tue, 20 Nov 2012 19:49:49 ESThttp://www.sciencedaily.com/releases/2012/11/121120194926.htmRibosome regulates viral protein synthesis, revealing potential therapeutic targethttp://www.sciencedaily.com/releases/2012/11/121120132906.htm Rather than target RNA viruses directly, aiming at the host cells they invade could hold promise, but any such strategy would have to be harmless to the host. Now, a surprising discovery made in ribosomes may point the way to fighting fatal viral infections such as rabies.Tue, 20 Nov 2012 13:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121120132906.htmHow does antibiotic resistance spread? Scientists find answers in the nosehttp://www.sciencedaily.com/releases/2012/11/121120121835.htm Microbiologists studying bacterial colonization in mice have discovered how the very rapid and efficient spread of antibiotic resistance works in the respiratory pathogen, Streptococcus pneumoniae (also known as the pneumococcus). The team found that resistance stems from the transfer of DNA between bacterial strains in biofilms in the nasopharynx, the area just behind the nose.Tue, 20 Nov 2012 12:18:18 ESThttp://www.sciencedaily.com/releases/2012/11/121120121835.htmScientists identify inhibitor of myelin formation in central nervous systemhttp://www.sciencedaily.com/releases/2012/11/121120100155.htm Scientists have discovered another molecule that plays an important role in regulating myelin formation in the central nervous system. Myelin promotes the conduction of nerve cell impulses by forming a sheath around their projections, the so-called axons, at specific locations -- acting like the plastic insulation around a power cord.Tue, 20 Nov 2012 10:01:01 ESThttp://www.sciencedaily.com/releases/2012/11/121120100155.htm'Obese but happy gene' challenges the common perception of link between depression and obesityhttp://www.sciencedaily.com/releases/2012/11/121120084725.htm Researchers have discovered new genetic evidence about why some people are happier than others. The scientists have uncovered evidence that the gene FTO -- the major genetic contributor to obesity -- is associated with an eight per cent reduction in the risk of depression. In other words, it's not just an obesity gene but a "happy gene" as well.Tue, 20 Nov 2012 08:47:47 ESThttp://www.sciencedaily.com/releases/2012/11/121120084725.htmTelomere lengths predict life expectancy in the wild, research showshttp://www.sciencedaily.com/releases/2012/11/121119213144.htm Researchers have found that biological age and life expectancy can be predicted by measuring an individual's DNA. They studied the length of chromosome caps -- known as telomeres -- in a 320-strong wild population of Seychelles Warblers on a small isolated island.Mon, 19 Nov 2012 21:31:31 ESThttp://www.sciencedaily.com/releases/2012/11/121119213144.htmCancer: Some cells don't know when to stophttp://www.sciencedaily.com/releases/2012/11/121119171403.htm Certain mutated cells keep trying to replicate their DNA -- with disastrous results -- even after medications rob them of the raw materials to do so, according to new research.Mon, 19 Nov 2012 17:14:14 ESThttp://www.sciencedaily.com/releases/2012/11/121119171403.htmMultiple sclerosis ?immune exchange? between brain and blood is uncoveredhttp://www.sciencedaily.com/releases/2012/11/121119163301.htm DNA sequences obtained from a handful of patients with multiple sclerosis have revealed the existence of an ?immune exchange? that allows the disease-causing cells to move in and out of the brain.Mon, 19 Nov 2012 16:33:33 ESThttp://www.sciencedaily.com/releases/2012/11/121119163301.htmFruit fly studies guide investigators to molecular mechanism frequently misregulated in human cancershttp://www.sciencedaily.com/releases/2012/11/121119132056.htm Changes in how DNA interacts with histones ?- the proteins that package DNA ?- regulate many fundamental cell activities from stem cells maturing into a specific body cell type or blood cells becoming leukemic. These interactions are governed by a biochemical tug of war between repressors and activators, which chemically modify histones signaling them to clamp down tighter on DNA or move aside and allow a gene to be expressed.Mon, 19 Nov 2012 13:20:20 ESThttp://www.sciencedaily.com/releases/2012/11/121119132056.htm3-D light switch for the brain: Device may help treat Parkinson's, epilepsy; aid understanding of consciousnesshttp://www.sciencedaily.com/releases/2012/11/121119114249.htm A new tool for neuroscientists delivers a thousand pinpricks of light to individual neurons in the brain. The new 3-D "light switch", created by biologists and engineers, could one day be used as a neural prosthesis that could treat conditions such as Parkinson's and epilepsy by using gene therapy to turn individual brain cells on and off with light.Mon, 19 Nov 2012 11:42:42 ESThttp://www.sciencedaily.com/releases/2012/11/121119114249.htmNew factor of genetic susceptibility to Alzheimer's diseasehttp://www.sciencedaily.com/releases/2012/11/121119104944.htm A large-scale international study has just discovered a gene for susceptibility to a rare disease providing evidence of the heterogeneous aetiology of Alzheimer's disease.Mon, 19 Nov 2012 10:49:49 ESThttp://www.sciencedaily.com/releases/2012/11/121119104944.htmBlood cancer gene BCL6 identified as a key factor for differentiation of nerve cells of cerebral cortexhttp://www.sciencedaily.com/releases/2012/11/121119093848.htm The cerebral cortex is the most complex structure in our brain and the seat of consciousness, emotion, motor control and language. In order to fulfill these functions, it is composed of a diverse array of nerve cells, called cortical neurons, which are affected by many neurological and neuropsychiatric diseases. Researchers have opened new perspectives on brain development and stem cell neurobiology by discovering a gene called BCL6 as a key factor in the generation of cortical neurons during embryonic brain development.Mon, 19 Nov 2012 09:38:38 ESThttp://www.sciencedaily.com/releases/2012/11/121119093848.htmMinority report: Insight into subtle genomic differences among our own cellshttp://www.sciencedaily.com/releases/2012/11/121118141530.htm Scientists have demonstrated that induced pluripotent stem cells -- the embryonic-stem-cell look-alikes whose discovery a few years ago won this year's Nobel Prize in medicine -- are not as genetically unstable as was thought.Sun, 18 Nov 2012 14:15:15 ESThttp://www.sciencedaily.com/releases/2012/11/121118141530.htmSkin cells reveal DNA's genetic mosaichttp://www.sciencedaily.com/releases/2012/11/121118141524.htm The prevailing wisdom has been that every cell in the body contains identical DNA. However, a new study of stem cells derived from the skin has found that genetic variations are widespread in the body's tissues, a finding with profound implications for genetic screening.Sun, 18 Nov 2012 14:15:15 ESThttp://www.sciencedaily.com/releases/2012/11/121118141524.htmLikely basis of birth defect causing premature skull closure in infants identifiedhttp://www.sciencedaily.com/releases/2012/11/121118141432.htm Geneticists, pediatricians, surgeons and epidemiologists have identified two areas of the human genome associated with the most common form of non-syndromic craniosynostosis premature closure of the bony plates of the skull.Sun, 18 Nov 2012 14:14:14 ESThttp://www.sciencedaily.com/releases/2012/11/121118141432.htmDNA packaging discovery reveals principles by which CRC mutations may cause cancerhttp://www.sciencedaily.com/releases/2012/11/121117184658.htm A new discovery concerning a fundamental understanding about how DNA works will produce a "180-degree change in focus" for researchers who study how gene packaging regulates gene activity, including genes that cause cancer and other diseases.Sat, 17 Nov 2012 18:46:46 ESThttp://www.sciencedaily.com/releases/2012/11/121117184658.htmHepatitis C treatment's side effects can now be studied in the labhttp://www.sciencedaily.com/releases/2012/11/121116161059.htm Adverse side effects of certain hepatitis C medications can now be replicated in the lab, thanks to a research team. The new method aids understanding of recent failures of hepatitis C antiviral drugs in some patients, and could help to identify medications that eliminate adverse effects. The findings may aid the development of safer and more effective treatments for hepatitis C and other pathogens such as SARS and West Nile virus.Fri, 16 Nov 2012 16:10:10 ESThttp://www.sciencedaily.com/releases/2012/11/121116161059.htmReconsidering cancer's bad guyhttp://www.sciencedaily.com/releases/2012/11/121116124644.htm Researchers have found that a protein, known for causing cancer cells to spread around the body, is also one of the molecules that trigger repair processes in the brain.Fri, 16 Nov 2012 12:46:46 ESThttp://www.sciencedaily.com/releases/2012/11/121116124644.htmGene distinguishes early birds from night owls and helps predict time of deathhttp://www.sciencedaily.com/releases/2012/11/121116124551.htm New research shows that a gene is responsible for a person's tendency to be an early riser or night owl -- and helps determine the time of day a person is most likely to die.Fri, 16 Nov 2012 12:45:45 ESThttp://www.sciencedaily.com/releases/2012/11/121116124551.htmClues to cause of kids' brain tumorshttp://www.sciencedaily.com/releases/2012/11/121116091226.htm Insights from a genetic condition that causes brain cancer are helping scientists better understand the most common type of brain tumor in children.Fri, 16 Nov 2012 09:12:12 ESThttp://www.sciencedaily.com/releases/2012/11/121116091226.htmArthritis study reveals why gender bias is all in the geneshttp://www.sciencedaily.com/releases/2012/11/121115210541.htm Researchers have pieced together new genetic clues to the arthritis puzzle in a study that brings potential treatments closer to reality and could also provide insights into why more women than men succumb to the disabling condition.Thu, 15 Nov 2012 21:05:05 ESThttp://www.sciencedaily.com/releases/2012/11/121115210541.htmClass of RNA molecules protects germ cells from damagehttp://www.sciencedaily.com/releases/2012/11/121115172255.htm Passing one's genes on to the next generation is a mark of evolutionary success. So it makes sense that the body would work to ensure that the genes the next generation inherits are exact replicas of the originals. Biologists have now identified one way the body does exactly that.Thu, 15 Nov 2012 17:22:22 ESThttp://www.sciencedaily.com/releases/2012/11/121115172255.htmQuick test speeds search for Alzheimer's drugs: Compound restores motor function and longevity to fruit flieshttp://www.sciencedaily.com/releases/2012/11/121115152655.htm Researchers report that an efficient, high-volume technique for testing potential drug treatments for Alzheimer's disease uncovered an organic compound that restored motor function and longevity to fruit flies with the disease.Thu, 15 Nov 2012 15:26:26 ESThttp://www.sciencedaily.com/releases/2012/11/121115152655.htmProtein-making machinery can switch gears with a small structural change process; Implications for immunity and cancer therapyhttp://www.sciencedaily.com/releases/2012/11/121115133414.htm For the past several years, research has focused on the intricate actions of an ancient family of catalytic enzymes that play a key role in translation, the process of producing proteins. In a new study, scientists have shown that this enzyme can actually also work in another fundamental process in humans.Thu, 15 Nov 2012 13:34:34 ESThttp://www.sciencedaily.com/releases/2012/11/121115133414.htmPlant derivative, tanshinones, protects against sepsis, study suggestshttp://www.sciencedaily.com/releases/2012/11/121115133312.htm Researchers have discovered that tanshinones, which come from the plant Danshen and are highly valued in Chinese traditional medicine, protect against the life-threatening condition sepsis.Thu, 15 Nov 2012 13:33:33 ESThttp://www.sciencedaily.com/releases/2012/11/121115133312.htmStructure of enzyme topoisomerase II alpha unravelled providing basis for more accurate design of chemotherapeutic drugshttp://www.sciencedaily.com/releases/2012/11/121115132903.htm Medical researchers have for the first time described the structure of the active site core of topoisomerase II alpha, an important target for anti-cancer drugs. The type II topoisomerases are important enzymes that are involved in maintaining the structure of DNA and chromosome segregation during both replication and transcription of DNA. One of these enzymes, topoisomerase II alpha, is involved in the replication of DNA and cell proliferation, and is highly expressed in rapidly dividing cancer cells.Thu, 15 Nov 2012 13:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121115132903.htmNewly discovered enzyme important in the spreading of cancerhttp://www.sciencedaily.com/releases/2012/11/121115132901.htm Enzyme hunters at UiO have discovered the function of an enzyme that is important in the spreading of cancer. Cancer researchers now hope to inhibit the enzyme.Thu, 15 Nov 2012 13:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121115132901.htmGenetics point to serious pregnancy complication, pre-eclampsiahttp://www.sciencedaily.com/releases/2012/11/121115132613.htm New research has revealed a genetic link in pregnant moms - and their male partners - to pre-eclampsia, a life-threatening complication during pregnancy.Thu, 15 Nov 2012 13:26:26 ESThttp://www.sciencedaily.com/releases/2012/11/121115132613.htmMolecular mechanisms underlying stem cell reprogramming decodedhttp://www.sciencedaily.com/releases/2012/11/121115132344.htm Thanks to some careful detective work, scientist better understand just how iPS cells form ? and why the Yamanaka process is inefficient, an important step to work out for regenerative medicine. The findings uncover cellular impediments to iPS cell development that, if overcome, could dramatically improve the efficiency and speed of iPS cell generation.Thu, 15 Nov 2012 13:23:23 ESThttp://www.sciencedaily.com/releases/2012/11/121115132344.htmSurprising genetic link between kidney defects and neurodevelopmental disorders in kidshttp://www.sciencedaily.com/releases/2012/11/121115132342.htm About 10 percent of kids born with kidney defects have large alterations in their genomes known to be linked with neurodevelopmental delay and mental illness, a new study has shown.Thu, 15 Nov 2012 13:23:23 ESThttp://www.sciencedaily.com/releases/2012/11/121115132342.htmEven moderate drinking in pregnancy can affect a child's IQhttp://www.sciencedaily.com/releases/2012/11/121114172833.htm Relatively small levels of exposure to alcohol while in the womb can influence a child's IQ, according to a new study using data from over 4,000 mothers and their children.Wed, 14 Nov 2012 17:28:28 ESThttp://www.sciencedaily.com/releases/2012/11/121114172833.htmGene nearly triples risk of Alzheimer's, international research team findshttp://www.sciencedaily.com/releases/2012/11/121114171710.htm A gene so powerful it nearly triples the risk of Alzheimer's disease has been discovered by an international team of researchers. It is the most potent genetic risk factor for Alzheimer's identified in the past 20 years.Wed, 14 Nov 2012 17:17:17 ESThttp://www.sciencedaily.com/releases/2012/11/121114171710.htmDiscovery could lead to faster diagnosis for some chronic fatigue syndrome caseshttp://www.sciencedaily.com/releases/2012/11/121114171708.htm For the first time, researchers have landed on a potential diagnostic method to identify at least a subset of patients with chronic fatigue syndrome - testing for antibodies linked to latent Epstein-Barr virus reactivation.Wed, 14 Nov 2012 17:17:17 ESThttp://www.sciencedaily.com/releases/2012/11/121114171708.htmResearch breakthrough could halt melanoma metastasis, study suggestshttp://www.sciencedaily.com/releases/2012/11/121114153227.htm In laboratory experiments, scientists have eliminated metastasis, the spread of cancer from the original tumor to other parts of the body, in melanoma by inhibiting a protein known as melanoma differentiation associated gene-9 (mda-9)/syntenin.Wed, 14 Nov 2012 15:32:32 ESThttp://www.sciencedaily.com/releases/2012/11/121114153227.htmPig genomes provide massive amount of genomic data for human healthhttp://www.sciencedaily.com/releases/2012/11/121114134512.htm Researchers provide a whole-genome sequence and analysis of number of pig breeds, including a miniature pig that serves a model for human medical studies and therapeutic drug testing.Wed, 14 Nov 2012 13:45:45 ESThttp://www.sciencedaily.com/releases/2012/11/121114134512.htm

Source: http://www.sciencedaily.com/rss/health_medicine/genes.xml

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Wednesday, November 28, 2012

Japan's new Restoration Party seeks bigger defense spending

TOKYO (Reuters) - A new Japanese party that hopes to become a force to contend with in a December 16 general election is calling for more defense spending to protect national interests and lower corporate and income taxes to bolster the economy, domestic media said on Thursday.

The Japan Restoration Party, which came in second to the main opposition Liberal Democrats in an opinion poll published on Thursday, also wants to shrink the role of the central government while promoting free-market competition and making it easier to revise Japan's pacifist constitution.

The party's platform, to be unveiled later on Thursday, calls for breaking through a decades-old unofficial cap that limited defense spending to 1 percent of gross domestic product and boosting maritime surveillance, the Yomiuri newspaper said. The moves that could further strain ties with China, already frayed by a feud over islands in the East China Sea.

About 15 percent of voters surveyed by the Nikkei business daily plan to vote for the Japan Restoration Party, outstripping the ruling Democratic Party of Japan's (DPJ) 13 percent but lagging behind the Liberal Democratic Party (LDP) at 23 percent.

Echoing LDP pledges of aggressive monetary easing to fight deflation, the Restoration Party wants to revise a law governing the Bank of Japan law - a move critics worry would lessen central bank independence.

The party, which had dropped its anti-nuclear stance after its founder, popular Osaka Mayor Toru Hashimoto decided to merge with a tiny pro-nuclear party led by the former governor of Tokyo, Shintaro Ishihara, will promise to "phase out" existing reactors by the 2030s to reduce reliance on nuclear power.

Public opposition to nuclear power in Japan has increased since the Fukushima radiation crisis last year, the world's worst in a quarter century.

(Reporting by Antoni Slodkowski; Editing by Linda Sieg)

Source: http://news.yahoo.com/japans-restoration-party-seeks-bigger-defense-spending-030615611.html

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President Obama has chosen Elise Walter to temporarily head the Securities and Exchange Commission (SEC) when Mary Schapiro steps down next month.? With Mary Schapiro leaving and Walter, a current commissioner stepping up, the SEC will be run by only one acting chairman and two other commissioners.? This means that the already delayed rule making for both the Dodd-Frank Wall Street Reform Act and the Jumpstart our Business Startups Act (JOBS) are likely to be further delayed.

The already taxed SEC will be headed by a small and temporary team.? Accordingly, it is the popular belief among experts that rule making will continue at a snail?s pace and that no major reform or policy changes should be expected.

Ms. Walter was originally appointed to the SEC by President George W. Bush in 2008 and has worked alongside Mary Schapiro both at the SEC and FINRA prior to both joining the SEC.? Moreover, Walter served as acting chairman after the departure of Christopher Cos in 2009, and so is familiar with the position.? She will stay in office until December 2013 when Obama will need to nominate a permanent new chief of the regulatory body.

The Author

Attorney Laura Anthony,
Founding Partner, Legal & Compliance, LLC
Securities, Reverse Merger and Corporate Attorneys

Securities attorney Laura Anthony provides ongoing corporate counsel to small and mid-size public companies as well as private companies intending to go public on the over-the-counter market, including the OTCBB and OTCQB. For nearly two decades, Ms. Anthony has dedicated her securities law practice to being ?the big firm alternative.? Clients receive fast and efficient cutting-edge legal service without the inherent delays and unnecessary expense of ?partner-heavy? securities law firms.

Ms. Anthony?s focus includes, but is not limited to, crowdfunding, registration statements, PIPE transactions, private placements, reverse mergers, and compliance with the reporting requirements of the Securities Exchange Act of 1934, including Forms 10-Q, 10-K and 8-K, as well as the proxy requirements of Section 14. Moreover, Ms. Anthony represents both target and acquiring companies in reverse mergers and forward mergers, including the preparation of deal documents such as Merger Agreements, Stock Purchase Agreements, Asset Purchase Agreements and Reorganization Agreements. Ms. Anthony prepares the necessary documentation and assists in completing the requirements of federal and state securities laws and SROs such as FINRA and DTC for corporate changes such as name changes, reverse and forward splits and change of domicile.

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Source: http://securities-law-blog.com/2012/11/27/mary-schapiro-to-be-replaced-by-elisse-walter-as-head-of-sec/

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Senate Dems divided over cuts to benefit programs

(AP) ? Deep divisions among Senate Democrats over whether cuts to popular benefit programs like Medicare and Medicaid should be part of a plan to slow the government's mushrooming debt pose a big obstacle to a deal for avoiding a potentially economy-crushing "fiscal cliff," even if Republicans agree to raise taxes.

Much of the focus during negotiations seeking an alternative to $671 billion in automatic tax increases and spending cuts beginning in January has centered on whether Republicans would agree to raising taxes on the wealthy. President Barack Obama has insisted repeatedly that tax increases on the wealthy must be part of any deal, even as White House officials concede that government benefit programs will have to be in the package too.

"It is the president's position that when we're talking about a broad, balanced approach to dealing with our fiscal challenges, that that includes dealing with entitlements," White House press secretary Jay Carney said Tuesday.

But even if GOP lawmakers agree to raise taxes, there is no guarantee Democrats can come up with enough votes in the Senate to cut benefit programs ? as Republicans are demanding.

"I hope not if it means Social Security or Medicare benefit cuts," said Sen. Sheldon Whitehouse, D-R.I.

There's a growing consensus among Senate Democrats and the White House that Social Security should be exempt from any deficit-reduction package. But some centrist Democrats in the Senate argue that fellow Democrats must be willing to consider cuts to Medicare and Medicaid in order to get concessions from Republicans on taxes.

"It has to be both ? a significant revenue increase as well as spending cuts," said Sen. Max Baucus, D-Mont., chairman of the Senate Finance Committee.

Sen. Kent Conrad, D-N.D., who is retiring as chairman of the Senate Budget Committee, said rising health care costs in Medicare and Medicaid are helping to drive future spending, making them an essential part of a long-term deficit-reduction package.

"I've been part of every bipartisan group here. We've always put everything on the table," Conrad said. "If you're going to solve this problem, you're going to have to deal with where the spending is and the revenue can be raised."

But senators like Baucus and Conrad increasingly are being drowned out by other Democrats emboldened by the recent election results to fight against benefit cuts.

Sen. Tom Harkin, D-Iowa, said he is willing to find savings in Medicare and Medicaid by making them more efficient. But, he said, he won't support benefit cuts.

"I think the election spoke very strongly about the fact that the vast majority of American people don't want to cut these programs," Harkin said.

Congress and the White House are devoting the next three weeks to finding at least a bridge over the fiscal cliff by reducing the sudden jolt of higher taxes and spending cuts in January while laying a framework for addressing the nation's long-term financial problems next year.

Obama wants to let tax rates rise for wealthy families while sparing middle- and low-income taxpayers. Some Republican leaders, including House Speaker John Boehner of Ohio, have said they were willing to consider making the wealthy pay more by reducing their tax breaks. But most Republicans in Congress adamantly oppose raising anyone's tax rates.

Negotiations are going slowly as each side waits for the other to make concessions.

Democrats already have tried to take Social Security off the table. Carney, the White House spokesman, said Monday that changes to the massive retirement and disability program should be done separately from any plan to reduce the deficit. That's the same position taken by 28 Democratic senators and independent Sen. Bernie Sanders of Vermont in a letter to fellow senators in September.

"We will oppose including Social Security cuts for future or current beneficiaries in any deficit-reduction package," said the letter, which was signed by many top Democrats, including Majority Leader Harry Reid of Nevada. In the House, Democratic leader Nancy Pelosi has taken the same position, not only on Social Security, but also on Medicare and Medicaid.

"There hasn't been the slightest suggestion about what they're going to do about the real problems, and that's entitlements," said Sen. Orrin Hatch of Utah, the top Republican on the Senate Finance Committee. "There's a certain cockiness that I've seen that is really astounding to me since we're basically in the same position we were before" the election.

The No. 2 Senate Democrat, Dick Durbin of Illinois, has tried to find common ground, saying he would be willing to require wealthy seniors to pay more for Medicare benefits. But on Tuesday, he said those discussions shouldn't be part of rushed negotiations to avoid the fiscal cliff.

"If we simply stand by and say 'don't touch Medicare in any way, for any reason, ever' we are inviting a crisis that opponents can exploit to eviscerate Medicare or even to end it," Durbin said. "Progressives should be willing to talk about ways to ensure the long-term viability of Social Security, Medicare and Medicaid, but those conversations should not be part of a plan to avert the fiscal cliff."

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Follow Stephen Ohlemacher on Twitter: http://twitter.com/stephenatap

Associated Press

Source: http://hosted2.ap.org/APDEFAULT/3d281c11a96b4ad082fe88aa0db04305/Article_2012-11-28-Fiscal%20Cliff-Entitlements/id-3bef0f5c28f446afb292115de0e000f1

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Simon Cowell Responds to Martin Gore, Labels Singer "Stupid" for Gun Remarks

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